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They provide a frameworkfor the developingT cells; thus, they correspond to the reticu-lar cells and their associated reticular fibers in other lymphatictissues and organs. Some theorists focus on a structural–functional framework that, when applied to family assessment,examines the interaction between the family and its internaland external environment (Friedman, 2002). These sections will discuss: 1) the use or induction of non-cell-to-cell mechanisms,which make use of soluble mediators to inhibit T cell responses and 2) cell-to-cell contactmechanisms.

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Gonococcal infection was historically a relativelycommon etiology of septic arthritis in sexually active individuals; in the 1970s, Neisseriagonorrhoeae accounted for about two-thirds of septic arthritis and tenosynovitis cases inNorth America [25]. Each value, expressed as apercentage of dead cells in the whole population represents the mean (± s.d.) of 3-5 distinctexperiments

Each value, expressed as apercentage of dead cells in the whole population represents the mean (± s.d.) of 3-5 distinctexperiments. Consistent changes in intracranial pressure waveformmorphology induced by acute hypercapnic cerebral vasodilatation

Consistent changes in intracranial pressure waveformmorphology induced by acute hypercapnic cerebral vasodilatation.

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This higher magnification shows a cross-sectioned villus containing several larger blood vessels and its thin surface syncytiotrophoblast layer.X200. It is sometimes described as an inability to judgedistance or scale

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Foreign body infections due to Staphylococcus epidermi-dis. Cyclooxygenase-2inhibitors may increase risk of myocardial infarction,and thiazolinediones are associated with precipitationof heart failure (4)

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In contrast to action onautonomic nerve endings, receptor activationon motor nerve endings augments ACh release,probably because it is of the 1 subtype.

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